Craig S. Pikaard Research Interest

Research Interest


Epigenetic gene silencing induced by genetic hybridization: When two species are crossed, genes inherited from one parent are sometimes preferentially expressed in the hybrid. How are parental alleles discriminated and selected? We are addressing these questions through genetic and molecular analyses of nucleolar dominance, an epigenetic phenomenon in which only one parental set of ribosomal RNA (rRNA) genes is expressed in an inter-species hybrid. The phenomenon has been known for ~70 years and occurs in plants, insects and animals. We have shown that inactive rRNA genes are preferentially silenced through changes in chromatin involving increased DNA (cytosine) methylation and decreased histone acetylation. Current efforts are focused on defining the mechanisms that discriminate the genes inherited from each parent and lead to the establishment of nucleolar dominance. Cytosine hypomethylation mutants are being tested to explore whether DNA methylation is involved in establishment as well as enforcement of dominance. rRNA transgenes integrated at chromosomal sites outside the rRNA gene clusters (NORs) will help us determine if silencing acts on individual rRNA genes or the multi-megabase NORs. Other studies will examine NOR-euchromatin junctions acting as boundaries between transcriptionally active and silent chromosomal domains. Sequences within these regions may play roles in NOR silencing.

Control of RNA polymerase I transcription: We are purifying RNA polymerase I complexes (holoenzymes) from both plants and Xenopus. These large (~2 Mda) complexes contain all the proteins required for promoter-dependent rRNA gene transcription. The complexes also contain protein kinase and chromatin modifying enzymes. Priorities include identifying and cloning holoenzyme subunits and determining the role of holoenzyme-associated activities in rRNA gene regulation.

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