Kellerman, K. and Miller, K.G. (1992) An unconventional myosin heavy chain gene from Drosophila melanogaster. J. Cell Biol. 119: 823-834.
Postner, M., Miller, K.G., and Weischaus, E. (1992) Maternal effect mutations of the sponge locus provides insight into actin cytoskeletal rearrangements in Drosophila melanogaster embryos. J. Cell Biol. 119: 1205-1218.
Mermall, V., McNally, J.G. and Miller, K.G. (1994) Transport of cytoplasmic particles in living Drosophila embryos catalyzed by an unconventional myosin. Nature 369: 560-562.
Miller, K.G. and D.R. Kellogg (1994) Isolation of cytoskeletal proteins from Drosophila. Methods in Cell Biol. 44: 259-277.
Mermall, V. and Miller, K.G. (1995) The 95F unconventional myosin is required for proper organization of Drosophila syncytial blastoderm embyos. J. Cell Biol. 129: 1575-1588.
Miller, K.G. (1995) The role of the actin cytoskeleton in early Drosophila development, Curr. Top. Devel 31: 167-196.
Miller, K.G. and D.P. Kiehart (1995) Fly Division (minireview). J. Cell Biol. 131: 1-5.
Hopmann, R., Cooper, J., and Miller, K.G. (1996) Actin organization, Bristle morphology and viability are affected by actin capping protein mutations in Drosophila., J. Cell Biol. 133: 1293-1305.
Lantz, V.L. and Miller, K.G. (1998) A Class VI Unconventional myosin is associated with a homologue of a microtubule-binding protein, Cytoplasmic Linker Protein-170, in neurons and at the posterior pole of Drosphila embryos. J. Cell Biol. 140: 897-910.
Lantz, V.L., Clemens, S., and Miller, K.G. The actin cytoskeleton is required for maintenence of posterior pole plasm components. Submitted.
Manuscripts in Preparation
Wagner, C.R., Mahowald, A., and Miller, K.G. One of the two cytoplasmic actin isoforms, Act5c, is essential in Drosophila.
Wagner, C.R., Mahowald, A., and Miller, K.G. An essential gene in Drosophila is related to a plant fertility protein.
Hopmann, R. and Miller, K.G. Capping protein is required for regulation of actin assembly during oogenesis.
Hicks, J., Rogat, A., and Miller, K.G. Loss of expression of an uncoventional myosin (class VI) in the testes results in sterility.